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Two SYSUCC projects get support from SINF

Last updated :2017-07-03

Source: Sun Yat-sen University Cancer Center
Written by: Sun Yat-sen University Cancer Center
Edited by: Wang Dongmei

Recently, MD Anderson Cancer Center (MDACC) announced its list of collaborative research projects supported by the 2016 Sister Institution Network Fund (SINF). Two projects from Sun Yat-sen University Cancer Center (SYSUCC) have been selected. One by Associate Professor Ying Huang from the Department of Radiation Oncology and another by Associate Professor Hailing Tang from the Department of Experimental Research. They will work on their respective projects along with faculty members from MDACC.

The Sister Institution Network Fund is a Global Academic Program developed in 2010 by MDACC of the United States. The purpose is to create collaborative oncology research projects between MDACC and its sister institutions and other global partners in the fight against cancer. The projects require collaboration between at least one MD Anderson Cancer Center Faculty member and or a Principal Investigator from one or more of the 40 Sister Institutions, located in over 22 countries.

Each application is scored following the NIH review criteria. Last year, 61 applications were submitted and peer reviewed from a select MD Anderson committee managed by the Office of Research Administration, as well as a review by the SINF Program Committee (one representative from each Sister Institution).

The Global Academic Program has approved the funding of 21 new SINF projects. They represent a diverse portfolio of cancer fighting science and reflect the expertise of the combined network of experts.

These collaborative projects will continue to strengthen SYSUCC’s relationship with MD Anderson as well as with the other sister institutions in the fight against cancer.

The two SYSUCC projects selected for SINF funding:
Associate Professor Ying Huang: Study long non-coding RNA NBR2 in esophageal cancer
Associate Professor Hailing Tang: Identification of kinases that are involved in PARP inhibitor resistance